Viewpoint analysis: "People with insulin resistance who undergo resistance training will not be effective.". Is this statement correct?

Views: 1     Author: Site Editor     Publish Time: 2024-05-05      Origin: Site

Viewpoint analysis: "People with insulin resistance who undergo resistance training will not be effective.". Is this statement correct?

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Recently, I found that some people have fallen into a misconception: if a person has abnormal glucose metabolism (such as insulin resistance), can they still increase their muscles and improve their metabolism through resistance training? Some people believe that it is not possible because they believe that muscles cannot obtain sufficient nutrition under the premise of insulin resistance, so resistance training is ineffective for individuals with metabolic abnormalities. The person who said this clearly has a one-sided understanding of metabolism. To make this statement valid, there must be a major premise: there is only one way to transport nutrients to muscles, which is through the insulin mediated pathway.

But what is the reality? In my previous introduction to glucose transporter protein (GLUT), I mentioned that there is a special subtype (GLUT1 type) that is insulin independent, which means that this transporter protein can transport nutrients such as blood sugar into the cell without relying on insulin, even at low insulin levels. These types of transporters are widely present in various parts of the body, but their expression levels are highest in endothelial cells of tissues such as red blood cells and the blood-brain barrier in adults. The GLUT3 subtype can also transport glucose into cells at very low glucose levels.

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Therefore, we can see that in order to survive, the body has derived two mechanisms for introducing nutrients into the interior of cells, one requiring insulin participation and the other not. Returning to the previous question: In the case of relatively insufficient insulin (when insulin resistance occurs, the amount of insulin in the body is too high, but work efficiency significantly decreases), the body's absorption of energy from external nutrients is indeed weakened, but this does not mean that the body cannot obtain nutrients at all.

Resistance training can improve the insulin receptor sensitivity of trainees and restore insulin work efficiency to its original state. Moreover, due to the relationship between resistance training, the transport proteins on muscle cell membranes that should have been activated by insulin can now be activated without the need for insulin. Therefore, resistance training can improve insulin sensitivity and metabolic efficiency on one hand; hgh before and after face, On the other hand, it can also enable muscle tissue to achieve a higher level of energy absorption without relying on insulin. So it's not that insulin resistance patients receiving resistance training is ineffective, on the contrary, this group of people are most likely to achieve the maximum health benefits from resistance training.

Long term resistance training can further amplify the benefits of insulin receptor sensitivity, and while controlling diet, resistance training can also help improve muscle fat ratio and reduce excess fat, which is also an important method for restoring insulin sensitivity.

It is undeniable that the insulin pathway plays a crucial role in mediating tissue growth, differentiation, energy utilization, and storage. If it can maintain high metabolic efficiency, it will definitely be beneficial for trainers. However, temporary insulin metabolism disorders are not an excuse to stop training or play around. Muscles are the largest glucose processing tissue in the human body, and regular resistance training is a necessary long-term task for improving (or even reversing) metabolic syndrome

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